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When cells become resistant to insulin, the receptors on their surfaces designed to respond to insulin have begun to malfunction. It simply means that the receptors require more insulin to make them work properly in removing sugar from the blood. Whereas before they needed just a touch to lower it, now they need a continuous supply of excess insulin to keep blood sugar within normal range.

As time goes by the blood sugar rises and stays high longer after a high carbohydrate meal even though there has been a lot of insulin made to lower it. You need to keep in mind that if your doctor were to check your sugar at this stage it would appear to be completely normal. The major silent change taking place is the ever-growing quantity of insulin needed to keep it that way.

The liver is the first organ in the body that becomes resistant to insulin, then the muscles and then the fat. The insulin is what controls the making of sugar by the liver. The sugar that is in your body at any given time is the result of two different things, the sugar that you ingest from different foods and drinks and the sugar that is made by your liver.

The sugar floating around in your body at any one time is the result of two things, the sugar that you have eaten and how much sugar your liver has made. When you wake up in the morning it is more of a reflection of how much sugar your liver has made. If your liver is listening to insulin properly it won’t make much sugar in the middle of the night. If your liver is resistant, those brakes are lifted and your liver starts making a bunch of sugar so you wake up with a bunch of sugar.

After your liver stops responding to insulin your muscles will be next. The muscles are designed to burn sugar that is made by your liver. If they are not responding to insulin the will not know that they need to burn the sugar. If you haven’t noticed there is a vicious chain going on, your liver is making too much sugar and your muscles can’t burn it off. So now what is affected?

Well the fat cells become resistant, but not for a while. It is only after a while that they become resistant. It takes them longer. Liver first, muscle second, and then your fat cells.

So for a while your fat cells retain their sensitivity. What is the action of insulin on your fat cells? To store that fat. It takes sugar and it stores it as fat. So until your fat cells become resistant you get fat, and that is what you see. As people become more and more insulin resistant, they get fat and their weight goes up.

But eventually they plateau. They might plateau at three hundred pounds, two hundred and twenty pounds, one hundred and fifty pounds, but they will eventually plateau as the fat cells protect themselves and become insulin resistant.

As all these major tissues, this massive body becomes resistant, your liver, muscles and fat, your pancreas is putting out more insulin to compensate, so you are hyperinsulinemic [having an abnormally high level of insulin in the blood] and you’ve got insulin floating around all the time.

Insulin floating around in the blood causes a plaque build up. Insulin causes the blood to clot too readily. Insulin causes cells that accumulate fatty deposits. Every step of the way, insulin’s got its fingers in it and is causing cardiovascular disease. It fills it with plaque, it constricts the arteries, it increases platelet adhesiveness and ability of the blood to coagulate [clot]. Any known cause of cardiovascular disease, insulin is a part of.

If you want to know if insulin sensitivity can be restored to its original state, well, perhaps not to its original state, but you can restore it to the state of about a ten year old.

You can increase sensitivity by diet and a lot of supplements.